We are analyzing https://link.springer.com/article/10.1007/bf00296494.

Title:
Neurofibrillary tangle predominant form of senile dementia of Alzheimer type: a rare subtype in very old subjects | Acta Neuropathologica
Description:
Acta Neuropathologica - In a consecutive autopsy series of 580 demented elderly subject, 256 with the clinical diagnosis of probable/possible Alzheimer
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {📚}

Education
Science
Health & Fitness

Content Management System {📝}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month

Based on our best estimate, this website will receive around 5,000,016 visitors per month in the current month.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Link.springer.com Make Money? {💸}

We're unsure how the site profits.

While many websites aim to make money, others are created to share knowledge or showcase creativity. People build websites for various reasons. This could be one of them. Link.springer.com might be cashing in, but we can't detect the method they're using.

Keywords {🔍}

google, scholar, disease, alzheimers, alzheimer, neurol, neurofibrillary, dementia, senile, neuropathol, braak, tangles, acta, article, pathology, aging, jellinger, clinical, plaques, bancher, diagnosis, neuropathological, neurology, ann, type, cortex, assessment, price, neurobiol, van, privacy, cookies, content, subjects, cases, entorhinal, access, related, hyman, duyckaerts, terry, exp, data, publish, research, search, predominant, years, state, amygdala,

Topics {✒️}

nincds-adrda work group month download article/chapter glial tau-positive inclusions neurofibrillary tangle densities privacy choices/manage cookies �neurofibrillary predominant type abundant neurofibrillary tangles full article pdf van hoesen gw common pathological feature pathologic diagnostic criteria neocortical neurofibrillary tangles �mini-mental state” european economic area scope submit manuscript consecutive autopsy series differential early pathologic van hoeven kh lewy body variant wong-riley mtt paired helical filaments sex-related differences related dementing conditions tau precedes ubiquination basic research conditions privacy policy differential laminar distributions quantitative immunohistochemical analysis clinico-pathologic analysis memoryrelated neuronal systems clinico-pathological documentation β-a4 deposits alzheimer neurofibrillary tangles accepting optional cookies informant-based clinical related immnohistochemical markers βa4 amyloid protein interlaboratory histopathologic assessment regional synpatic pathology entorhinal cortex pathology rare subtype occurring entorhinal-hippocampal connections amygdala cell loss journal finder publish assist neuropathologic study alzheimer type characterized predominant involvement global brain disease neuritic ad pathology check access

Schema {🗺️}

WebPage:
      mainEntity:
         headline:Neurofibrillary tangle predominant form of senile dementia of Alzheimer type: a rare subtype in very old subjects
         description:In a consecutive autopsy series of 580 demented elderly subject, 256 with the clinical diagnosis of probable/possible Alzheimer's disease (AD), there were 10 cases aged between 80 and 99 years with moderate to severe dementia or confusional state in which neuropathological studies revealed abundant neurofibrillary tangles with predominant involvement of the allocortex (entorhinal region, subiculum, CA 1 sector of hippocampus, amygdala) but no or only very few senile plaques. Small numbers of diffuse deposits of βA4 amyloid protein were present in the entorhinal cortex of 3 and in the isocortex of 5 brains, while neuritic plaques were totally absent. Only a few cases of this “senile dementia with tangles only” or, more correctly, “neurofibrillary predominant type of AD” corresponding to the limbic stage of neuritic AD pathology have been described in the literature. This rare subtype occurring in very old (over 80 years of age) subjects that does not fall within the currently used neuropathological criteria for diagnosis of AD warrants further clinico-pathological documentation.
         datePublished:
         dateModified:
         pageStart:565
         pageEnd:570
         sameAs:https://doi.org/10.1007/BF00296494
         keywords:
            Dementia
            Alzheimer's disease
            Neurofibrillary tangles
            Limbic Alzheimer pathology
            Pathology
            Neurosciences
         image:
         isPartOf:
            name:Acta Neuropathologica
            issn:
               1432-0533
               0001-6322
            volumeNumber:88
            type:
               Periodical
               PublicationVolume
         publisher:
            name:Springer-Verlag
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
            type:Organization
         author:
               name:C. Bancher
               affiliation:
                     name:Lainz Hospital
                     address:
                        name:L. Bioltzmann Institute of Clinical Neurobiology, Lainz Hospital, Vienna, Austria
                        type:PostalAddress
                     type:Organization
               type:Person
               name:K. A. Jellinger
               affiliation:
                     name:Lainz Hospital
                     address:
                        name:L. Bioltzmann Institute of Clinical Neurobiology, Lainz Hospital, Vienna, Austria
                        type:PostalAddress
                     type:Organization
               type:Person
         isAccessibleForFree:
         hasPart:
            isAccessibleForFree:
            cssSelector:.main-content
            type:WebPageElement
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:Neurofibrillary tangle predominant form of senile dementia of Alzheimer type: a rare subtype in very old subjects
      description:In a consecutive autopsy series of 580 demented elderly subject, 256 with the clinical diagnosis of probable/possible Alzheimer's disease (AD), there were 10 cases aged between 80 and 99 years with moderate to severe dementia or confusional state in which neuropathological studies revealed abundant neurofibrillary tangles with predominant involvement of the allocortex (entorhinal region, subiculum, CA 1 sector of hippocampus, amygdala) but no or only very few senile plaques. Small numbers of diffuse deposits of βA4 amyloid protein were present in the entorhinal cortex of 3 and in the isocortex of 5 brains, while neuritic plaques were totally absent. Only a few cases of this “senile dementia with tangles only” or, more correctly, “neurofibrillary predominant type of AD” corresponding to the limbic stage of neuritic AD pathology have been described in the literature. This rare subtype occurring in very old (over 80 years of age) subjects that does not fall within the currently used neuropathological criteria for diagnosis of AD warrants further clinico-pathological documentation.
      datePublished:
      dateModified:
      pageStart:565
      pageEnd:570
      sameAs:https://doi.org/10.1007/BF00296494
      keywords:
         Dementia
         Alzheimer's disease
         Neurofibrillary tangles
         Limbic Alzheimer pathology
         Pathology
         Neurosciences
      image:
      isPartOf:
         name:Acta Neuropathologica
         issn:
            1432-0533
            0001-6322
         volumeNumber:88
         type:
            Periodical
            PublicationVolume
      publisher:
         name:Springer-Verlag
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:C. Bancher
            affiliation:
                  name:Lainz Hospital
                  address:
                     name:L. Bioltzmann Institute of Clinical Neurobiology, Lainz Hospital, Vienna, Austria
                     type:PostalAddress
                  type:Organization
            type:Person
            name:K. A. Jellinger
            affiliation:
                  name:Lainz Hospital
                  address:
                     name:L. Bioltzmann Institute of Clinical Neurobiology, Lainz Hospital, Vienna, Austria
                     type:PostalAddress
                  type:Organization
            type:Person
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
["Periodical","PublicationVolume"]:
      name:Acta Neuropathologica
      issn:
         1432-0533
         0001-6322
      volumeNumber:88
Organization:
      name:Springer-Verlag
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Lainz Hospital
      address:
         name:L. Bioltzmann Institute of Clinical Neurobiology, Lainz Hospital, Vienna, Austria
         type:PostalAddress
      name:Lainz Hospital
      address:
         name:L. Bioltzmann Institute of Clinical Neurobiology, Lainz Hospital, Vienna, Austria
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:C. Bancher
      affiliation:
            name:Lainz Hospital
            address:
               name:L. Bioltzmann Institute of Clinical Neurobiology, Lainz Hospital, Vienna, Austria
               type:PostalAddress
            type:Organization
      name:K. A. Jellinger
      affiliation:
            name:Lainz Hospital
            address:
               name:L. Bioltzmann Institute of Clinical Neurobiology, Lainz Hospital, Vienna, Austria
               type:PostalAddress
            type:Organization
PostalAddress:
      name:L. Bioltzmann Institute of Clinical Neurobiology, Lainz Hospital, Vienna, Austria
      name:L. Bioltzmann Institute of Clinical Neurobiology, Lainz Hospital, Vienna, Austria
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {🔗}(90)

Analytics and Tracking {📊}

Google Tag Manager

Libraries {📚}

Clipboard.js
Prism.js

CDN Services {📦}

Crossref

3.58s.

LINK . SPRINGER . COM {}