Here's how GENESDEV.CSHLP.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
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GENESDEV . CSHLP . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Genesdev.cshlp.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. External Links
  10. Analytics And Tracking
  11. Libraries

We are analyzing https://genesdev.cshlp.org/content/16/8/984.

Title:
p53 regulates cell survival by inhibiting PIK3CA in squamous cell carcinomas
Description:
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fields
Website Age:
17 years and 0 months (reg. 2008-06-12).

Matching Content Categories {๐Ÿ“š}

  • Science
  • Telecommunications
  • Education

Content Management System {๐Ÿ“}

What CMS is genesdev.cshlp.org built with?

Custom-built

No common CMS systems were detected on Genesdev.cshlp.org, and no known web development framework was identified.

Traffic Estimate {๐Ÿ“ˆ}

What is the average monthly size of genesdev.cshlp.org audience?

๐Ÿš„ Respectable Traffic: 10k - 20k visitors per month


Based on our best estimate, this website will receive around 10,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Genesdev.cshlp.org Make Money? {๐Ÿ’ธ}

We don't see any clear sign of profit-making.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Genesdev.cshlp.org has a revenue plan, but it's either invisible or we haven't found it.

Keywords {๐Ÿ”}

cell, pikca, laboratory, cancer, author, survival, york, pten, article, submit, carcinomas, usa, cells, pathways, apoptosis, tumors, email, cold, spring, harbor, press, home, subscribe, advertise, info, archive, contact, search, regulates, inhibiting, squamous, epithelial, biology, center, transcriptional, required, pmediated, prosurvival, pik, primary, ofp, malignant, inhibition, independent, neoplasms, fax, full, articles, issue, online,

Topics {โœ’๏ธ}

cell death/survival cell survival involves phosphorylated akt fraction diminishing prosurvival activities pi3k/aktpathways play p53-mediated apoptosis p53-related apoptosis ting chao chou2 pornchai o-charoenrat1 p110ฮฑ catalytic subunit upper aerodigestive tract mutually exclusive events ptenin epithelial tumors epithelial cancer biology org/cgi/doi/10 0890-9369 online issn article doi pten deficient cells article ยป table cancer center inhibiting pik3ca cancer biology carcinomas mutation ofp53 issue alert article benign cells primary tumors malignant cells pi3k andy goberdhan1 christine walsh3 su dao1 ivan ngai1 archontoula stoffel3 baylor college abstract interactions significant role transcriptional regulation oncogene involved simultaneous abnormalities malignant phenotype constitutive activation received december 31 accepted february 22 p53 pten exerts search rockefeller university transcriptional inhibition

Questions {โ“}

  • What's this?

Analytics and Tracking {๐Ÿ“Š}

  • Facebook Pixel
  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {๐Ÿ“š}

  • jQuery

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