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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Javascript Libraries
  13. Hosting Providers

We began analyzing https://www.nature.com/articles/ncb0400_241, but it redirected us to https://www.nature.com/articles/ncb0400_241. The analysis below is for the second page.

Title[redir]:
TRAIL receptor-2 signals apoptosis through FADD and caspase-8 | Nature Cell Biology
Description:
Nature Cell Biology - TRAIL receptor-2 signals apoptosis through FADD and caspase-8

Matching Content Categories {📚}

  • Education
  • Science
  • Telecommunications

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🏙️ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 76,749,346 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

We don’t know how the website earns money.

Many websites are intended to earn money, but some serve to share ideas or build connections. Websites exist for all kinds of purposes. This might be one of them. Doi.org might be cashing in, but we can't detect the method they're using.

Keywords {🔍}

article, nature, cas, google, scholar, caspase, cell, trailr, cells, fadd, death, trail, apoptosis, biol, access, science, content, receptor, schneider, biology, juo, receptors, fig, references, author, research, permissions, signals, bodmer, holler, reynard, blenis, jürg, tschopp, fas, ligand, binding, signalling, disc, bjab, shown, data, curr, rights, privacy, advertisement, journals, search, log, journal,

Topics {✒️}

nature portfolio permissions reprints nature 388 nature author information authors cell-surface receptor fas inhibiting t-cell activation springerlink instant access trail-receptor-induced apoptosis author correspondence anti-dr5 antibodies developed trail-receptor disc isolated caspase-8 jean-luc bodmer data programmed cell death escaping cell death called death receptor explore content subscription content mutant cell lines cell growth differ communication published trail-induced death apoptosis-independent role issue learn jürg tschopp cell biology trail induces apoptosis permissions journals search log cytoplasmic death domain studied disc assembly nat cell biol recently identified member produced controversial results identical signalling molecules institutional subscriptions read harvard medical school suppressing joint inflammation tumour-necrosis factor trail-r2 disc access trail decoy receptors https express trail-r2 trail-r2 bound activated trail-r2 pascal schneider peter juo genetic evidence10

Schema {🗺️}

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      name:Department of Cell Biology, Harvard Medical School , Massachusetts, USA
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Social Networks {👍}(1)

External Links {🔗}(201)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Tag Manager
  • Google Universal Analytics

Javascript Libraries {📚}

  • Prism.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com
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