Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We are analyzing https://www.ncbi.nlm.nih.gov/pmc/articles/PMC452261/.

Title:
A slow sustained increase in cytosolic Ca2+ levels mediates stalk gene induction by differentiation inducing factor in Dictyostelium. - PMC
Description:
During Dictyostelium stalk cell differentiation, cells vacuolate, synthesize a cellulose cell wall and die. This process of programmed cell death is accompanied by expression of the prestalk gene ecmB and induced by the differentiation inducing factor ...
Website Age:
26 years and 11 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Education
  • Telecommunications
  • Science

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🚀🌠 Tremendous Traffic: 10M - 20M visitors per month


Based on our best estimate, this website will receive around 10,880,080 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We see no obvious way the site makes money.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Ncbi.nlm.nih.gov might be making money, but it's not detectable how they're doing it.

Keywords {🔍}

pubmed, google, scholar, dictyostelium, cell, pmc, calcium, free, article, gene, differentiation, dif, discoideum, expression, intracellular, embo, cai, biochem, dec, aug, biol, nlm, cytosolic, stalk, cells, kay, development, nov, williams, cytoplasmic, oct, increase, levels, prestalk, ecmb, induced, increases, effects, page, references, biophys, gross, sci, feb, main, content, ncbi, search, sustained, induction,

Topics {✒️}

n'n'-tetra-acetic acid ng115-401l neuronal cells controlling cell-fate decisions camp-dependent protein kinase blocked dif-induced expression high-affinity ca2+ pump stalk gene expression cytosolic ca2+ concentration cytoplasmic free calcium main content prestalk gene ecmb jellyfish apoaequorin cdna intracellular ca2+ pump mobilizing intracellular ca2+ pmc disclaimer stalk cell differentiation intracellular calcium homeostasis ca2+-atpase inhibitor 2 mouse lymphoma cells cell lines expressing differentiation inducing factor differentiation-inducing factor free calcium ca2+ entry pathway inhibiting ca2+ efflux insensitive ca2+ stores atp-driven ca2+/ united states government messenger phosphoprotein res biochim biophys acta proton pumps populate cellular slime mold semi-synthetic aequorins cytoplasmic acidification facilitates glucocorticoid-induced lymphocytolysis cellulose cell wall programmed cell death exp cell res original print version bhq-induced [ca2+] pmc website suppresses cell growth methods cell biol complete article early eukaryote evolution arch biochem biophys morphogen controlling differentiation embo journal article image cytosolic ca2+

External Links {🔗}(55)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager
  • Google Universal Analytics

Libraries {📚}

  • FontAwesome
  • jQuery
  • Popper.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov
  • ns.nih.gov
  • ns2.nih.gov
  • ns3.nih.gov

CDN Services {📦}

  • https://maxcdn.bootstrapcdn.com

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